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health health health health health health health We bred our first litter in 1989, after several years of
studying, joining Akita clubs in England (where we lived until 1987) and in
the USA, then eventually purchasing our dogs (one at a time!), studying some
more, showing them to their championships, studying, talking, listening,
attending seminars, studying pedigrees, doing health checks, studying…
and eventually we bred our first litter. Frankly, my opinion is that no one
is qualified to breed for the first several litters, because experience is
one of the best teachers and you obviously don’t start out with
experience, lol! We were no
different, but I think we had beginners luck in many respects. We were
plagued by longcoats initially, but had no actual health problems for several
years. And our beautiful coated puppies, with the exception of our beloved
Tigger who we kept, were placed in pet homes with spay/neuter agreements,
which of course is required for all pets. Our
first actual health problems started in
2001. A dog we had bred was diagnosed with SA at the age of five. This was truly devastating; I had only vaguely
heard of this disease, and read frightening bits about it. I didn’t
even think I knew anyone that had produced it, let alone imagined one of our
own dogs would ever get it. It came
from a breeding of our lovely Scarlet to an outside dog (Nia Minda Catcher In
The It turned out that despite
denials by Catcher’s owner, Catcher has produced SA with other bitches,
all unrelated to ours. He has also produced pemphigus and lupus. His
mother’s full sister has produced VKH and SA, and Catcher’s own
father, owned by the same people that own Catcher, produced SA, so it seems
that some of this tragedy could have been avoided with a little bit of
honesty. Curiously, in spite of full knowledge of these facts,
Catcher’s owners continue to breed him, fouling the Although we have never
produced SA in any of our breedings except the one to Catcher, we acknowledge
that it is believed by experts to be a simple recessive, meaning that both
parents had to be carriers. That is why we spayed Scarlet immediately –
she was a carrier. Looking back, it is most likely Scarlet got the gene from
her father, Joey, since the disease has surfaced in Joey’s background.
Apparently Joey himself was never bred to an SA carrier, since he never
produced it, but you can see how the gene can be passed on from one
generation to the next by carriers, and all you need is two carriers and
you’ve got SA. This situation continues
to haunt us as we move from one generation to the next. The only positive aspect of this tragedy is
that we have been provided a map of where we We will continue to add any other problems we produce to
this list in the hopes that the information provided may be of help to
someone else. If you have specific questions, please feel free to contact us.
We believe that honesty can and will be the salvation of our wonderful breed. |
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© Copyright 1999-2008, Hoka-Hey
Akitas. All rights reserved. |
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