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health health health health health health health We bred our first litter in 1989, after several years of
studying, joining Akita clubs in England (where we lived until 1987) and in
the USA, then eventually purchasing our dogs (one at a time!), studying some
more, showing them to their championships, studying, talking, listening,
attending seminars, studying pedigrees, doing health checks, studying…
and eventually we bred our first litter. Frankly, my opinion is that no one
is qualified to breed for the first several litters, because experience is
one of the best teachers and you obviously don’t start out with
experience! A real Catch 22
situation! We were no different, but
I think we had beginners luck in many respects. We were plagued by longcoats
initially, but had no actual health problems for several years. And our
beautiful coated puppies, with the exception of our beloved Tigger who we
kept, were placed in pet homes with spay/neuter agreements, which of course
is required for all pets. Our
first actual health problem was in
2001. A dog we had bred was diagnosed with SA at the age of five. This was truly devastating; I had only
vaguely heard of this disease, and read frightening bits about it. I
didn’t even think I knew anyone that had produced it, let alone
imagined one of our own dogs would ever get it. It came from a breeding of our lovely
Scarlet to an outside dog that was totally unrelated to ours – Nia Minda Catcher In
The Rye. There were eight puppies
in that litter. Besides the one with Comanche lived to over 14
years old and was one of our most adored house pets. All seven of Comanche’s siblings were
also neutered because of the diagnosis of SA in their littermate, Ryder, showing
a great deal of courage and ethics on the part of their owners. It was sad for us to see that Catcher’s
owners, having proof he was an SA carrier (and he actually had produced it
before our litter with Scarlet, which we only found out later from those
litter owners), he was continually used at stud. This is exactly how our gene
pool is fouled. Although we have never
produced SA in any of our breedings except the one, we acknowledge that it is
accepted by experts as a simple recessive gene, meaning that both parents
have to be carriers to produce it. That is why we spayed Scarlet immediately
– she was a carrier. Her parents are Joey and Sealy. Looking back, it is almost certain Scarlet
got the gene from her father Joey, since there are multiple reports of ‘skin
problems’ in Joey’s background. Apparently Joey himself was never
bred to an SA carrier, since he never produced it, but you can see how the
gene is passed on from one generation to the next by carriers, and all you
need is two carriers somewhere down the line and you’ve got SA in your
litter. This situation continues
to haunt us as we move from one generation to the next. We will do our best to make this kind of
information available to the public in an effort to spare anyone else the
heartbreak that goes with the diagnosis of such an insidious disease in a
beloved family member. We will continue to add any genetic diseases we produce to
this list, in the hopes that the information provided may be of help to
someone else. We were blindsided when it happened to one of our dogs, and
that made it even harder. If you have specific questions, please feel free to
contact us. We believe that honesty can and will be the salvation of our
wonderful breed. |
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© Copyright 1999-2011, Hoka-Hey Akitas. All rights
reserved. |
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